Internal Jugular Phlebectasia in a Patient with Facial Trauma

injury: a randomized, controlled, experimental study. Crit Care 2013;17:R86. 5. Robson MC, Del Beccaro EJ, Heggers JP. The effect of prostaglandins on the dermal microcirculation after burning, and the inhibition of the effect by specific pharmacological agents. Plast Reconstr Surg 1979;63:781-7. and the left MCA contraction period blood flow velocity was 98 cm/sec, which was higher than the normal MCA contraction period blood flow velocity of approximately 60 cm/sec. To diagnose cerebral vasospasm, the blood flow velocity from the TCD should be above 120 cm/sec. As the test was performed three days after the injury, it could be assumed that cerebral vasospasm had been improving. Cardiacorigin embolus can occur in patients with atrial fibrillation or cardiac valve disease. However, in this case, the electrocardiography and echocardiography were normal. Therefore, the cause of cerebral infarction was not a cardiac-origin embolus. In addition, based on the entry and exit of the current pathway, the cause of brain damage was not direct electrical injury. However, we cannot completely exclude cerebral infarction caused by necrotic emboli, which is similar to MRI findings, since there are no applicable tests to determine whether the cause of cerebral infarction is necrotic emboli. Nevertheless, based on examinations and comprehensive tests, we believe that the cause of cerebral infarction was vasospasm rather than necrotic emboli. In the present case, we experienced cerebral infarction caused by high-voltage electrical injury. Accordingly, active evaluation was needed for brain injury in the high-voltage electrical injury. In addition, thorough examinations and comprehensive tests were critical in the treatment of various complications, as they can be easy to miss because of severe external injuries by high-voltage electrical injury. With a review of the relevant literature, this paper has discussed a case of cerebral infarction caused by cerebral vasospasm in a 44-year-old male patient who suffered from high-voltage electrical injury from contact with 22,900 V.